Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. Then again, our capability to rescue SERT perform in KI midbrain synaptoneurosomes from the inhibition of FAK implies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by increased pFAK localization in 5-HT synapses. Our https://reidboyju.ka-blogs.com/85594304/pro33-options