Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our capability to rescue SERT function in KI midbrain synaptoneurosomes by the inhibition of FAK implies elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by increased pFAK localization in five-HT synapses. https://englandh319kxh2.blogoxo.com/profile